Home
This site is intended for healthcare professionals
Advertisement

Trauma Series: Knee Trauma | Fahad Hossain

Share
Advertisement
Advertisement
 
 
 

Summary

Attend this informative session to learn about the assessment and management of knee dislocations. The faculty for the session is Fahad Siddique Hossain, a Consultant Sports and Arthroplasty Knee Surgeon and Director of Research and Development at Walsall Healthcare NHS Trust. He will cover all aspects of management related to this injury, from recent literature to potential for ischemia and limb loss to the epidemiology, aetiology, classification, clinical assessment, vascular assessment, nerve injury, initial management, definitive management, and a summary.

Generated by MedBot

Description

An MP3 of this talk can be found here - feedback form to be used for slides

Introduction:

Dr. Fahad Siddique Hossain, a Consultant Sports and Arthroplasty Knee Surgeon from the Walsall Healthcare NHS Trust in the UK, provides an in-depth review on the management of knee dislocations, focusing on the assessment and management during the initial period of orthopaedic consultation and discussing recent literature concerning definitive management.

Objectives:

  1. Understand the potential risks of ischaemia and limb loss associated with knee dislocations.
  2. Analyze the data regarding popliteal artery injury and its relation to amputation rates in knee dislocation.

Epidemiology:

  • Knee dislocations are extremely rare, constituting 2-29 per million orthopaedic injuries.
  • Up to half may self-reduce before a formal assessment.
  • The condition is more common among younger individuals, with a 4:1 ratio in males to females.

Aetiology:

  • High Energy Injuries: Account for 50% of knee dislocations and include causes like crush injuries and road traffic accidents.
  • Low Energy Injuries: One-third of dislocations arise from sports injuries and falls.
  • Ultra Low Energy Injuries: Those with a BMI greater than 48 during normal daily activities.

Classification:

Based on Kennedy et al. 1963, knee dislocations are categorized as:

  • Anterior
  • Posterior
  • Lateral
  • Medial
  • Rotatory (anteromedial and anterolateral)

Clinical Assessment:

  • Begin with the ATLS protocol, particularly for high energy or multiple trauma cases.
  • Then, focus on the knee to ensure perfusion, reduction, and stabilization.
  • Take note of gross deformities, medial skin puckering, and invaginated capsule. Swelling and haemoarthrosis might not always be evident.

Vascular Injury:

  • 40% may have a concomitant arterial injury.
  • Signs include pallor, coolness, pulsatile hematoma, delayed capillary refill, and absent distal pulses.

Vascular Assessment:

  • The arteriogram remains the gold standard. However, modern non-invasive techniques such as CT Arteriogram are preferred.
  • The ideal time for an arteriogram after a knee dislocation remains debated.

Nerve Injury:

  • Occurs in up to 40% of cases.
  • The most commonly affected nerve is the common peroneal nerve, mainly due to traction injuries.
  • In cases of foot drop, an orthosis is needed. If there's no improvement by the time of definitive treatment, surgery might be necessary.

Initial Management:

  • Often, the knee can be reduced in a closed manner.
  • Up to 50% present post spontaneous reduction.
  • The focus should be to immobilize, then reassess.

Definitive Management:

  • Surgical intervention outperforms non-surgical treatment in terms of recovery scores and the rate of return to sports or work.
  • Repair vs. Reconstruction remains a debated topic, but early intervention (within 3 weeks) yields better outcomes.
  • No significant difference in outcomes was noted between early and late surgery for severe injuries, but staged surgical treatment often offers the best functional outcomes.

Conclusion:

Knee dislocations are rare yet critical injuries that require a high index of suspicion due to the high rate of associated neurovascular complications. Immediate treatment should follow the ATLS protocol, aiming to reduce, re-perfuse, and reassess before planning definitive management.

Learning objectives

Learning Objectives:

  1. Understand the epidemiology and aetiology of knee dislocation.
  2. Recognize the classification of knee dislocation, according to Kennedy et al. (1963).
  3. Develop an understanding of the vascular complications associated with knee dislocation.
  4. Learn the principles of initial management for knee dislocation, including reduction and immobilization.
  5. Gain insight into the surgical management of knee dislocations, including indications and timing.
Generated by MedBot

Similar communities

View all

Similar events and on demand videos

Advertisement
 
 
 
                
                

Computer generated transcript

Warning!
The following transcript was generated automatically from the content and has not been checked or corrected manually.

Management of knee dislocations Fahad Siddique Hossain Consultant Sports and Arthroplasty Knee Surgeon Director of Research and Development Walsall Healthcare NHS Trust United Kingdom• No conflicts of interest Objectives • Review assessment and management for day one orthopaedic consultant • Discuss recent literature on definitive management• Potential for ischaemia and limb loss independent of mechanism and energy • Average rate of popliteal artery injury 16% (1.6 to 40%) • 20% amputation rate associated with vascular injury in knee dislocation – LEAP study Epidemiology • Extremely rare! – 2-29 per million orthopaedic injuries • Up to 50% self reduce before formal assessment • Predominantly young population – Male:Female 4:1 Aetiology High Energy Injuries • – 50 % of all knee dislocations – Crush injuries, RTA etc • Low Energy Injuries – Third of knee dislocations – Sports injuries and falls • Ultra low Energy Injuries – BMI greater than 48 – Normal ADLs Classification • Kennedy et al. 1963 – Anterior – Posterior – Lateral Medial – – Rotatory (anteromedial and anterolateral) • Difficult to apply in clinical situationSchenk et al. 1999 Clinical Assessment • ATLS protocol in the setting high energy/multiple trauma • Then focus on knee – Ensure perfusion, reduction and stabilization – Grossly deformed – Medial puckering of skin • Invaginated capsule – Swelling and haemoarthrosis not always present Vascular Injury Up to 40% may have concomitant arterial injury • Hard signs – Pallor, coolness – Pulsatile haematoma Delayed capillary refill – – Absent distal pulses semitendinosus 3 2 1 8 Biceps Semimembranosus femoris 9 10 Lat. Gastroc Med. Gastroc 4 7 5 11 6 Vascular Assessment • Arteriogram is gold standard – Modern non invasive methods such as CT Arteriogram • No consensus on indications or when after knee dislocation • In all casesvs cases with hard signs of ischaemia Current Thinking! • Latent period of vascular injury – Non – flow limiting arterial lesion in acute post reduction period – Delayed progression of intimal tear – Secondary ischaemia from thrombosisSelective Arteriography Protocol Clin J Sport Med 2009;19 [2]:125-129 Nerve Injury • Up to 40% of cases –Common peroneal nerve most common –Usually traction related injury Nerve Injury • Up to 40% of cases –Common peroneal nerve most common –Usually traction related injury • Suspect LCL and PLC injury– Foot drop requiring orthosis to prevent equinus contracture – Exploration and neurolysis if no improvement by time of definitive treatment – If no evidence of nerve continuity by 3 months than surgery Initial Management • Reduce (often closed) – Up to 50% present after spontaneous reduction • Immobilise • Reassess Irreducible joint • Buttonhole of MFC through MCL • Compromised medial skin with pinching • May require open reductionDefinitive Management• Surgery better than non operative treatment Improved Tegner, Lysholm and IKDC score after surgery – – Higher rate of return to sport and work • Repair vs Reconstruction – not clear! – Similar Lysholm and Tegner Scores – Higher failure rate in PLrepair – Better return to sport after reconstruction (similar rates of return to work)• Early surgical intervention better than delayed – Cut off 3 weeks • Tissue planes can be identified • Allows easy re-approximation and suture placement – Higher Tegener Lysholm and IKDC score with early surgery – Higher rate of return to sports• No difference between early and late surgery in Schenck Type 3+ injuries Staged surgical treatment yields best functional • outcomes (79.1% vs 58.4% with excellent or good scores) Summary • Rare Injury • High Index of suspicion • High rate of associated neurovascular injury • Initial treatment – ATLS – Reduce and re-perfuse – Restrict – Reassess • Plan definitive managementThank you