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Summary

Join the on-demand teaching session, ChildHealth: OrthopaedicsFracturesinPaediatricsDifferenceinPhysiology, to further your understanding of orthopaedics and fractures within pediatric physiology. This comprehensive course will delve into the structure and peculiarities of children’s bone makeup - from growth plate functions, bone types and fracture types to an in-depth look at the Salter-Harris classification. The session will cover diagnosis techniques, including imaging and blood tests, as well as management practices ranging from surgical and mechanical alignments to treatments for different Salter-Harris types. The course will conclude with discussions on conditions such as Irritable Hip, Septic Arthritis, and Idiopathic Juvenile Arthritis, among others. Don't miss this opportunity to unravel the complex interactions between physiology, pathophysiology and clinical management concerning orthopaedics in pediatrics.

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Learning objectives

  1. Understand and explain the differences in the physiology and structure of paediatric and adult bones, including growth (epiphyseal) plates and types of bone in children's bodies.
  2. Identify and describe various types of fractures, including Salter-Harris (Growth Plate) fractures, and their relevance to paediatric orthopaedics.
  3. Demonstrate knowledge in how to manage specific orthopaedic injuries, including fractures, the role of conservative, medical, surgical management and the specific considerations in paediatric patients.
  4. Recognise and manage conditions that commonly present in paediatric orthopaedics such as Transient Synovitis, Septic Arthritis and Juvenile Idiopathic Arthritis.
  5. Evaluate the challenges and special considerations related to paediatric orthopaedics, including cases of non-accidental injury, codeine and tramadol metabolism, and the prognosis of different conditions and injuries.
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ChildHealth: OrthopaedicsFracturesinPaediatricsDifferenceinPhysiology Growth (epiphyseal) Plates Hyaline cartilage between epiphysis and metaphysis Areas of bone allowing it to grow in length Not found in adults: These plates fuse during puberty, the remenants of which become epiphyseal linesDifferenceinPhysiology Bone type Children’s bones contain more cancellous bones -> are more spongey and vascularised Bones are more malleable/flexible Hence, able to form greenstick fractures, where one side breaks and the other side is intact Bones are less strong More likely to get a buckle/torus fracture following compression TypesofFractures Does anyone know ANY types of fractures (adult and paediatric)?TypesofFractures TypesofFractures Salter-Harris (Growth Plate) SALTR mnemonic Straight across Above BeLow Through CRush Investigations Bloods group and save Imaging X ray: rule of 2s 2 views 2 joints 2 occasions (compare with old images if availbale) CT/MRI: soft-tissue injuryManagement Conservative Consider safeguardingNon-AccidentalInjury Delayed presentation Developmental/Milestone delay Inconsistent history Multiple injuries Injuries at sites not exposed to traumaManagement Medical Pain managementAnalgesicstoAvoid Codeine and tramadol: Unpredictable metabolism Aspirin: Reye’s syndrome risk, avoid in asthmatics Exception: Kawasaki diseaseManagement Surgical Mechanical aligment Closed/open reduction Stability External casts K wires Intramedullary wires Intramedullary nails Screws Plate and screwsManagement Salter-Harris Type 1/2: usually minimally displaced closed reduction If 5+ day post-injury, do not attempt manipulation If poorly aligned: operative management immobilisation Type 3/4: greater displacement consider ORIF Type 5: referral to specialistsPrognosis Usually good If missed/mismanaged -> Disruption Early closure of growth plateIrritableHip Transient Synovitis Definition Irritation to synovial membrane Most common cause of hip pain in Often preceded by viral URTIPresentation Limp Avoiding weight bear Groin/hip pain Low grade temperature Note: If temperature and systemic signs, consider septic arthritis Management Safety net: if feverish, go to A&E Usually improves 1-2 weeksArthritis:SepticAetiology Staphylococcus aureus (most common) Neisseria gonorrhoea (gonococcus) in sexually active teenagers Group A streptococcus (Streptococcus pyogenes) Haemophilus influenza Escherichia coli (E. coli) Riskfactors Age (younger, <3) Pre-existing joint abnormalities Immunodeficient Recent trauma/surgery Skin infectionPresentation Joint pain with systemic signs Usually knee/hip Stiffness Reduced movement Systemic symptomsManagament Investigations Joint aspiration MC+S (before abx) raised WCC Imaging: USS, MRI ABCDE Abx asap initially IV (2 weeks) 4-6w oral Irrigation and debridement (washout)Arthritis:IdiopathicJuvenileIdiopathicArthritis Idiopathic arthritis seen in <16s lasting >6w Associated with HLA A2, HLA B27 Risk factors Female FHx (first degree) Down’s syndrome Subtype Joint involvement Other features Oligoarthritis (most common) 1-4 large joints RF negative polyarthritis 5+ large/small joints Symmetrical RF+ polyarthritis 5+ large/small joints Symmetrical h Fever spikes Salmon-colour rash Systemic-onset (Still’s) 1+ joints Generalised lymphadenopathy, hepatosplenomegaly, serositis Psoriatic arthritis Enthesis related 1+ weight bearing joints Sacroilitis Undifferentiated Does not fulfil any category OR fits 2+ categories Differentials Cancer/malignancy Septic arthritis Reactive arthritis Osteomyelitis Rheumatic fever SLEInvestigations Bloods FBC exclude malignancy and infection JIA: thrombocytosis, anaemia, leukocytosis RF Anti-CCP ANA: +ve more susceptible to uveitis HLA-B27: enthesitis-related JIAInvestigations Imaging XRay undetectable in early disease rule out fracture USS: abnormal in early stages MRI contrast enhanced: most-sensitive for soft tissue and bone erosions Check for uveitisModerate intensity exercises Management aerobics, flexibility, strengthening Corticosteroids NSAIDs severe/systemic JIAgement for Initial symptomatic treatment use shortest possible duration limit to 2w at a time consider calcium/vit D supplements if Intra-articular glucocorticoid injections long-term preferred initial treatment If all else fails: Biologics 2nd line: DMARDs TNFa inhibitors: etanercept, 1st line: Methotrexate adalimumab, infliximab Leflunomide IL6 inhibitors Sulfalazine selective B cell blockade JAKiOthercausesof limps PerthesDisease Idiopathic avascular necrosis of the femoral head Disrupted blood flow to the femur Idiopathic: No trigger Consider SUFE if triggered by trauma Classic presentation: 5-8 year old (primary school) boy with a limp and pain in the hip/groin Management: ConservativeSlipped Upper Femoral Epiphysis Head of femur slips along growth plate Classic presentation: Secondary school boys, catogorised as obese, following minor trauma Pain often disproportionate to trauma. Externally rotated hip, limited internal rotation. Management: SurgeryDevelopmentalDysplasia oftheHip Structural abnormality in hip during gestation -> instability in hip Risk factors: FHx, Breech presentation, Multiple pregnancy Picked up during the NIPE: Ortolani and Barlow Confirm with USS Manage ASAP: Easier to manage when young Pavlik Harness